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There are a number of drugs available for the treatment of impotence. They can be taken by mouth, injected into the side of the penis, or inserted into the male urethra. Impotence drugs should only be taken as prescribed by a physician. It is important to understand who can take these drugs safely and who should avoid them. Viagra, Cialis, and Levitra are the brand names of some commonly prescribed oral impotence drugs. They all act by relaxing the smooth muscles in the penis, thereby increasing blood flow into the penis during sexual stimulation. While Viagra has been used the longest and has the most safety data available, all three drugs are safe and comparable in efficacy. Cialis has the longest duration of action, up to 48 hours, and has been nicknamed the ‘weekender’. These drugs should never be taken with nitrates, a group of drugs used to treat heart disease, as drug interactions can result in a dangerous drop in blood pressure. There are a number of conditions in which taking any of these drugs is contraindicated. Some common side effects such as headaches, flushing, diarrhea, and a stuffy nose may be experienced with any of these drugs. Labels for all three drugs are currently being modified to add nonarteritic ischemic optic neuropathy, a condition in which blood flow to the optic nerve is blocked resulting in vision loss as a rare side effect. Alprostadil (Caverject), papaverine (Pavabid), and phentolamine (Regitine) can be injected into the side of the penis with a tiny needle. These drugs act by increasing blood flow into the penis, and their dosage can be varied depending on how long they are required to act. The MUSE, or medicated urethral system for erections, involves the application of a small pellet containing alprostadil into the tip of the urethra. do penile enlargement pills really work best penile enlargment penile enlargement technique do pnis enlargement pills work enlargement forum free matter penis size penis enlargement before and after picture herbal penile enlargment pills penis enlargement

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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. 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Signs and Symptoms of Acne Rosacea There are certain contrasts between acne rosacea and acne vulgaris. As you probably know, acne vulgaris is the most common form of acne which seems to start with the onset of puberty and is characterized by the formation of whiteheads and blackheads. Acne rosacea, on the other hand, usually makes its first appearance between the ages of 30 and 50, is confined to the face -- mostly the nose, cheeks, chin, forehead and eyelids -- and is not associated with overactive oil glands. The lesions seen in acne rosacea consist of erythema, minute dilated blood vessels, papules and pustules. The color of the skin in the affected areas varies from bright to dull red, or it can even take on a purplish hue. At first the redness may last for just a few hours, but later as the condition progresses and recurrences continue, the color persists and can become permanent. The hypertrophy of the sebaceous glands, especially on the nose, leads to thickening of the skin, increased visibility of the expanded follicles and an enlargement of the nose, so often associated with acne rosacea. Although most commonly seen in women, men have the most severe cases and are the ones that usually develop the disfiguring bulbous nose, known as rhinophyma. Oddly, sometimes rhinophyma is the only sign of the condition. People suffering from acne rosacea come to realize what aggravates their particular outbreaks and avoid them, if possible. The most common stimulants are hot, spicy foods, hot beverages and alcohol. Exposure to the sun and to heat also seems to be aggravating factors. Acne rosacea is not life-threatening, but it certainly does severely alter a person's appearance. Complications Associated With Rosacea There are few complications associated with acne rosacea. Sometimes, eye irritations can occur due to inflammation of the eyelashes or outer surface of the eyes. The membrane covering the lens (cornea) can become inflamed leading to impaired vision, but this rarely happens. Treatment Options for Acne Rosacea Topical and Oral Medication. There is no proven cure for rosacea since the exact cause of the disease is not known. Successful treatment is based on controlling the acne-like symptoms with the same topical and systemic medications used in treating regular acne. The most effective treatment to date includes long term use of topical and oral antibiotics such as tetracycline which does seem to control the eruptions. The dosage of the antibiotic is slowly lowered to maintain control. In most cases, it can eventually be discontinued altogether without recurrence of the rosacea pimples. Of course, tetracycline should never be taken during pregnancy since the medication does affect the unborn child. Laser and Surgery. Laser treatment has been successful in eliminating the enlarged facial blood vessels. This is a treatment that causes very little discomfort. Also, surgery can be used to remove the excess tissue associated with rhinophyma. Other Treatment. The psychological and stress problems associated with acne rosacea should always be assessed and treated. cheapest penile enlargement pills vimax testimonials do penis enhancement pills work vimax penis enlargement forum penile enlargment surgery photo where to buy vigrx penis enhancement before and after picture natural pennis enlargement technique penile enlargement patch

Increased sexual pleasure has been a goal common to both men and women throughout history. However, achieving such a goal is not always as easy as one would like to think. While we commonly see depictions of intensely sensual sexual activities, hear about climatic orgasmic experiences, and can read about extremely long love-making sessions; bringing this flavor of enjoyment into our actual lives is not always easy as these media outlets make it seem. Some men might have problems achieving a hard erection and women often find they have a decreased sexual appetite. What them, can be done to achieve the type of sexual experiences that we currently only fantasize about? After intense research into this dilemma, it has been discovered that the intake of a high level of amino acids is of great assistance to redeveloping sexual desire and performance. For men, amino acids work to escalate the amount of ejaculation fluid which results not only in increased sperm count, but also greatly amplifies the sexual experience through prolonged orgasms. Not only is the penis able to increase in size and hardness, but stamina and desire is magnified. For women, amino acids allow a larger amount of blood to flow into their clitoris and vaginal tissues. This results in heightened sensitivity, which encourages sexual desire and activity. In addition, when a woman is taking amino acid supplements, her orgasms are extremely enhanced, leading to a drastic increase in sexual pleasure. One of the most popular amino acids on the market is L-Arginine, a semi-essential amino acid. It has been proven to create increased blood flow to the sexual organs of both men and women. L-Arginine is described as a free-form amino acid, one that is crystalline. Not only has L-Arginine been shown to improve sexual quality and performance, it is also known to work in relocating nitrogen throughout the body. It is also highly important to the body’s metabolism and is known to be beneficial to the activity of the blood vessels surrounding the heart. L-Arginine can be found in numerous sexual enhancement products. When looking for an L-Arginine supplement, try to find one that uses natural herbs in addition to high levels of amino acids. The combination results in increased stimulus for women, and penis enlargement and increased fluid ejaculation for men. In addition, L-Arginine has been proven to aid the body in many other ways, such as lowering blood pressure levels and is an overall healthy supplement to one’s health regime. truth about pennis enlargement vimax penis enlargement pills product penis enlargment video free penis enlargment video penile enlargement testimonials penis enlargement review plus vigrx do penis enlagement pills really work penile enlargement patch

Here you will learn all about misconceptions about penis size and how you can be perfectly happy with the size of penis you have. There are many misconceptions about penis size and here we separate fact from fiction. Currently penis size is big business there is more than $1 billion per year spent on vitamins, creams, pumps etc No one knows penis size better than condom manufacturers. Here are some facts on penis size from their research: •Research shows that more than 70 percent of men have penises that measure between 5 and 7 inches when erect. A penis is considered abnormally small only if it measures less than 3 inches when erect, and even then it might not matter •The above size statistics are true for all the races of mankind. The concept that black men have a large penis while Japanese have small ones is totally false. •A recent Dutch study of 375 sexually active women who had recently given birth suggests that the surveys are accurate — to a point. Seventy-nine percent of the women said that penis length is unimportant, and 69 percent of them said the same about penis girth. •Exercise and food supplements can enlarge the size of your penis is totally false. Penis size and your partner Your partner is the best judge of size, but try to remember when you were young and in high school. .. To make money, and feed on men’s insecurity about their penis size and possibility they may have a small penis many products are sold that simply dont work. How to increase small penis 4 methods guaranteed not to work! There are several methods recommended to increase penis size and surprise, surprise, none of them work. Here are 4 examples. 1.Stretching, and squeezing - sometimes known as jelquing. These are exercises that are supposed to be performed each day for an indefinite period of time. Although they appear safer than other methods, they can lead to severe scar formation, and disfigurement. The only result gained is a soft penis that has a somewhat larger appearance. This method has no affect on the erect penis. 2.Vacuum pumps and other devices. Because pumps force blood into the penis, making it swell, they can sometimes be useful in the treatment of impotence. This may create an illusion of a larger penis, and the results are seldom permanent. Rather, repeated damages the elastic tissue in the penis, finally producing softer erections, or no erections at all. 3.Stretching with penile weights. This technique is dangerous, and very likely to cause permanent damage to your penis. 4.Pills, lotions, and hormone creams. These can contain vitamins, minerals, herbs. They sometimes have chemicals similar to Viagra and Cialis. They also can contain hormones such as testosterone. Note that there is no evidence that it or any other substance can increase penis size in adult men. The opposite is rather the case. Another method to increase a small penis size is cosmetic surgery. In these surgical procedures, please note that EVERY professional medical society have all issued policy statements against cosmetic surgical procedures to enhance the penis. The results can be catastrophic. To lengthen a penis, the surgery typically involves cutting the suspensory ligament that attaches the penis to the pubic bone and moving skin from the abdomen to the penile shaft. As the suspensory ligament stabilizes and gives an upward tilt to an erect penis, after surgery, the penis will not longer be able to achieve this tilt. Such surgery can cause the penis to wobble, erect at strange angles, and more often than not, cause unpleasant side-effects. On the negative side, after various types of cosmetic penile enhancement surgery, some men have had to undergo additional correction operations to address the deformities caused by the original procedure. Some of the negative effects of this type of surgery are scarring, fat clumps, low-hanging penis, loss of sensitivity, bumps, lumps, shorter penis, and hair on the base of the penis. Other complaints include persistent pain, impotence, and urinary incontinence So, unless your partner (s) remarks that something is wrong, follow the old adage of “not fixing what isn’t broken”. On the other hand, take some time to understand your partner's physical, emotional needs and desires, and you may do a great deal more to improve your sexual relationship than would changing the size of your penis. The myth of the small penis is exactly that and most women are more interested in a loving partner more than penis size. Of course, there are exceptions but generally men should not be concerned over size.